TRIAL: A test to determine whether the species will create new organ designs to accommodate chronic over feeding or fail and result in mortality and long term decline. The alternate endpoint, increased intelligence in feeding strategies.
The test subjects all like sugar but dietary advice mostly says give it up. The question, what really happens when they eat excess sugar over time?
OK, but sugar is glucose and it is known that glucose is a fundamental fuel so what is wrong with that? Let's take a walk down bio-chemistry lane. Just a peak so easy peasy. This is really important if one has concerns about livers so don't run away yet.
Fructose’s propensity to cause fatty liver is unique among carbohydrates. The fatty liver directly causes insulin resistance setting in motion the vicious cycle of hyperinsulinemia – insulin resistance. Furthermore, this harmful effect of fructose does not require high blood glucose or blood insulin levels to wreak havoc. Further, this fattening effect, because it acts through fatty liver and insulin resistance, cannot be seen in the short term – only in the long term.
Fructose overconsumption directly produces fatty liver, which in turn directly creates insulin resistance. Fructose is five to ten times more likely than glucose to cause fatty liver. This sets off a vicious cycle. Insulin resistance leads to hyperinsulinemia, to ‘overcome’ this resistance. However, this backfires, as the hyperinsulinemia, made worse by the attendant glucose load, leads to further insulin resistance.
Watch this short video to learn about why fructose is bad for you to consume
This discussion focuses on fructose and why it is a real enemy of your liver so think high fructose corn syrup. The little secret is that table sugar is 50% fructose so it is not just calories that we are concerned about.